@proceedings {7695,
	title = {La douleur et la morphinoth{\'e}rapie},
	journal = {Journ{\'e}es de concertation pour la finalisation du programme national de Recherche en Sant{\'e}. 26-27 F{\'e}vrier },
	year = {1997},
	address = {Alger, Alg{\'e}rie},
	author = {Ounassa Adjroud}
}
@article {7660,
	title = {Les opiac{\'e}s endog{\`e}nes et la motricit{\'e} ut{\'e}rine pendant la gestation},
	journal = {Journal Alg{\'e}rien de M{\'e}decine },
	volume = {VII},
	number = {4},
	year = {1997},
	pages = {128-133},
	author = {Ounassa Adjroud}
}
@article {7659,
	title = {Action Of Naloxone And Acetorphan, On Uterine Contractions Induced By Oxytocin -in Periparturient Rat In Vivo},
	journal = {Synth{\`e}se},
	volume = {3},
	number = {1},
	year = {1997},
	pages = {63-68},
	abstract = {
	Theffects{\textquoteright} in vivo of acetorphan, an _enkephalinase inhibitor and naloxone, an opiate antagonist on the - - oxytocin- induced myometrial mechanical activity were studied at day 21 of pregnancy in rats. Acetarpban" I 10 mg kg -1 (i.v.) increased (344\%) spontaneous uterine contractions duration only at 0-10 min period, but - did not modify amplitude. This increase in the duration was blocked by naloxone 5 mg ks -1, which had no effect on the amplitude or duration of contractions when given alone. Oxytocin (~T), I, 10 and I 100 mIU (i.v.) stimulated in a dose dependent manner, spontaneous uterine contractions duration but did not alter the amplitude of contractions. Blocking the opioid peptides by naloxone, 5 mg kg -1 (s.c.) injected --10min before OT strongly potentiated the effect of the lowest dose of OT (1 mlU) b-yabout of 59\&amp;Ofn and doubled the amplitude of contractions during the 30 min observation period. When acetorphan, 10 mg kg _-1 (i.v.) administered 30 min before the lowest dose of OT studied, 1 mID (i.v.), It induced a slight increase by .about 88\% in uterine contractions duration produced by OT alone, The amplitude remained unchanged. These -results suggested that uterine contractions induced by OT in late pregnancy in rats might be regulated by enkephalinergie and enkephalinasic systems

},
	url = {https://www.asjp.cerist.dz/en/article/78944},
	author = {Ounassa Adjroud}
}
